Further developing the links between biological warfare research and the Lyme Disease establishment, we review information from FTR #585.
At every turn, Lyme disease research is inextricably linked with biological warfare research. Divided into the “Steere” and “ILADS” camps, the Lyme disease research community is split between the view that the disease is “hard-to-catch, easy-to-cure” and the diametrically opposed view that the disease is very serious and produces long-term neurological disorder. The Steere camp diminishes the significance of the disease and is closely identified with biological warfare research. At the epicenter of Lyme disease research (and the Steere camp) are members of the Epidemic Intelligence Service, or EIS. EIS personnel are to be found at every bend in the road of Lyme disease research.
The Borrelia genus has long been researched as a biological warfare vector. Note that Unit 731 personnel and their files were put to work for the United States after World War II, much like the Project Paperclip scientists from Germany. ” . . . borrelia were known for their ability to adopt different forms under conditions of stress (such as exposure to antibiotics). Shedding their outer wall, (which is the target of penicillin and related drugs), they could ward off attack and continue to exist in the body. . .”
Much of the program is devoted to excerpting and analysis of a 2013 posting by Elena Cook. This discussion of “Spirochete Warfare,” in turn, makes liberal use of material from a 1944 text about Japan’s biological warfare program. This book “Japan’s Secret Weapon,” contains a great deal of information about Japanese pioneering of the use of spirochetes as biological warfare organisms.
This material is to be considered in the historical and political context of the incorporation of the key personnel and files of the notorious Japanese Unit 731 biological warfare division into the U.S. BW program after World War II.
Apparently decades ahead of their Allied counterparts, Japanese use of spirochetes encompassed a number of important points to consider.
1.–The Japanese understood that “cell-wall deficient spirochetes, ” “granule” and “L-forms” had tremendous significance for biological warfare. ” . . . This WW2-era book helps to confirm what some investigating the history of Lyme disease have long suspected; that the official denial of the devastating pathogenic nature of the granule and other ‘L-forms'(1) of Lyme-causing Borrelia, is related to their biological warfare significance. . .”
2.–” . . . To put it bluntly, Newman’s book provides cogent circumstantial evidence that many Cell-wall deficient forms of Borrelia are in fact weaponized spirochetes, nurtured, cultured and optimized for aerosol delivery. . .”
3.–According to author Barclay Newman, a combined Japanese and Nazi biological warfare offensive against Hawaii using the spirochetal disease leptospirosis against Hawaii two or three years before the attack on Pearl Harbor: ” . . . . ‘Nazi and Japanese scientists cooperated in warfare against or with spirochetes – in Hawaii.’ (original author’s italics). What he is referring to is an exceptionally virulent outbreak of the spirochetal disease leptospirosis, also known as Weil’s disease, and known at the time in Germany as ‘slime fever’. With official reports of 44% mortality from the outbreak, Newman states: Consult the authorities, and you will find out that, very definitely, so high a mortality is attained only by Japanese strains of spirochetes of slime fever. . . .”
4.–According to Newman, the Japanese had concluded that spirochetes, although very close to bacteria in form, were not actually bacteria and therefore: ” . . . . a spirochete can also break itself into many tiny granules, each as small as the invisible molecule of a virus, and each capable of recreating a new spirochete. . . .”
5.–Again, according to Newman: ” . . . The Japanese have reported that you can increase the virulence, or killing power, of these spirals by growing them in flesh and blood, of guinea pig or man. . .” This is interesting to consider in light of the evidence of Lyme Disease as the product of biological warfare. Might some of the “tests” have had the goal of “growing” such organisms in humans? ” . . . The resistance of many spirochetes, including borrelia, to culture in vitro remains a problem for lab scientists even today. . .”
6.–The “granule” spirochete form was found by the Japanese to have great value for aerosolized BW applications: ” . . . Inada has reported that the Japanese know how to get virus-like, quite invisible particles or spirochete-fragments from special cultures of spirochetes of infectious jaundice. The Japanese say that such infinitesimals can be used to infect animals and men, by spraying droplets containing these spirochete-creating bits into the air, or spreading them through water, or scattering them in mud or damp soil. . . .”
7.–The above-mentioned leptospirosis or “slime fever” may have been used as a “softening-up” agent prior to Japanese invasions in World War II” ” . . . ‘Immediately before the Japanese invasions of China, Indo-China, the Dutch East Indies, and the Malay States, and shortly before the Japanese invasion of India and the Japanese strokes at Australia, the very first outbreaks of slime fever were reported from every one of these areas’ . . .”
8.–The Japanese had discovered the application of infection via multiple pathogens. This may have figured into the development of Lyme Disease as well. ” . . . Fujimori (sic) was testing out the effects of spreading two different parasites into the same guinea pig at the same time. The Japanese discovered that one parasite promotes the lethal action of the other. . . .”
9.–The Japanese developed with spreading spirochetal disease via spraying droplets into the eyes of targets. We wonder if Willy Burgdorfer’s possible Lyme infection from diseased Rabbit-urine may have stemmed from this technology? This is discussed below. ” . . . ‘Sometimes the Japanese think up the damnedest experiments, such as the transmission of syphilis by spraying the spirochetes into the air or into the eyes of animals or volunteers. Infection is thus accomplished. . . . if you want to speculate further about the possibilities of spirochete warfare, you can be sure that the Japanese know how to spread any spirochete disease . . . by spraying droplets laden with specially cultured spirochetes. . . .”
10.-Among the diseases apparently harnessed for BW use by the Japanese was African relapsing fever. Willy Burgdorfer did his graduate thesis about this tick-borne spirochetal disease and it was researched at length by his mentor Rudolf Geigy. (Geigy’s possible role as an I.G. Farben intelligence agent and Paperclip recruiter is discussed in FTR #1135. Note that some forms of Borrelia Burgdorferi–a primary causative agent of Lyme Disease–resemble the spirochete that causes relapsing fever. ” . . . Relapsing fever is caused by the Borrelia genus of bacteria, and is generally transmitted to man either by lice, or by the bite of a tick. It is worth noting, too, that recent investigations into the genetic make-up of Lyme borrelia have found some strains apparently more closely related to relapsing fever Borrelia than to Borrelia burgdorferi, long considered the only borrelia capable of causing Lyme disease. . . .”
Next, the program details Rudolf Geigy’s work on relapsing fever. We suspect that his interest in such afflictions was not as benign and altruistic as his defenders maintain. As mentioned above, Lyme Disease “discoverer” and biological warfare veteran Willy Burgdorfer did his graduate thesis on relapsing fever.
Again, as mentioned above, Willy Burgdorfer contracted what he felt was Lyme Disease after urine from an infected rabbit splashed into his eyes. We wonder if some of the techniques of using aerosolized spirochete granules might have been involved in Willy’s accidental infection? ” . . . .While he was rinsing off one of the trays in the sink, Lyme-infected rabbit urine splashed into his eyes. A few weeks later, on April 13, he noticed five Lyme bull’s-eye rashes under his armpit and on his torso. . . .”
In an unpublished manuscript, Willy Burgdorfer noted not only the persistence of Lyme Disease but its ability to remain dormant in the nervous system: “. . . . It is now clear that Borrelia burgdorferi can persist within the nervous system for years, causing progressive illness, and increasing evidence suggests also that the spirochete can remain latent there for years before producing clinical symptoms. . . .”
Lyme disease is difficult to diagnose, another factor that makes it ideal for BW use. Might the Japanese Unit 731 research into spirochetal warfare described by Barclay Newman have figured into some of the boiler-plate research that went into the development of Lyme Disease? ” . . . Lyme’s ability to evade detection on routine medical tests, its myriad presentations which can baffle doctors by mimicking 100 different diseases, its amazing abilities to evade the immune system and antibiotic treatment, would make it an attractive choice to bioweaponeers looking for an incapacitating agent. Lyme’s abilities as ‘the great imitator’ might mean that an attack could be misinterpreted as simply a rise in the incidence of different, naturally-occurring diseases. . . .”
There is experimental evidence that infection with Borrelia burgdorferi can produce the amyloid plaques symptomatic of Alzheimer’s Disease. ” . . . Here is hypothesized a truly revolutionary notion that rounded cystic forms of Borrelia burgdorferi are the root cause of the rounded structures called plaques in the Alzheimer brain. Rounded “plaques’ in high density in brain tissue are emblematic of Alzheimer’s disease (AD). . . .”
The program concludes with more experimental evidence of the production of amyloid deposits characteristic of Alzheimer’s Disease: ” . . . To determine whether an analogous host reaction to that occurring in AD could be induced by infectious agents, we exposed mammalian glial and neuronal cells in vitro to Borrelia burgdorferi spirochetes . . . Morphological changes analogous to the amyloid deposits of AD brain were observed following 2–8 weeks of exposure to the spirochetes. . . These observations indicate that, by exposure to bacteria or to their toxic products, host responses similar in nature to those observed in AD may be induced. . . .”